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Targeted keywords and phrases were selected for their relevance in assessing perioperative risks associated with GLP-1 receptor agonists along with appropriate Boolean operators and search filters

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And in many cases, they interact with endocrine, vascular, neurologic, or melanocortin pathways we still do not fully understand outside therapeutic contexts

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The rationale is that glucagon-receptor agonism increases energy expenditure and promotes hepatic lipid mobilization, effectively adding a burn component on top of the eat less component supplied by GLP-1