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Because the BPC-157 peptide operates through VEGFR2 and NO pathways that are mechanistically distinct from TB-500s actin-based signaling and GHK-Cus collagen synthesis mechanisms, the three compounds address complementary aspects of tissue repair without documented antagonism in animal studies

This would seem to be a potentially fruitful area for further research since the major environmental risk factors for PDexposure to pesticides, trichloroethylene, and air pollutionare likely to have their effects on dopaminergic neurodegeneration via toxic effects of oxidative stress and inflammation 61

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doi: 10.5555/URI:PII:0022214367900765 48 PandayS.TalrejaR.KavdiaM

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