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Abstract This study has been initiated to investigate whether sunitinib (SUN) alters the expression of key genes engaged in mitochondrial transport and oxidation of long chain fatty acids (LCFA), and if so, whether these alterations should be viewed as a mechanism of SUN-induced cardiotoxicity, and to explore the molecular mechanisms whereby carnitine supplementation could attenuate SUN-induced cardiotoxicity
Mitophagy receptor FUNDC1 regulates mitochondrial homeostasis and protects the heart from I/R injury
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Furthermore, both in vitro and in vivo , GLP-1 has been frequently demonstrated to reduce GI muscle activity via nerve-mediated mechanisms reliant on nitric oxide (53)
Regular follow-up monitoring is described as what keeps the approach both safe and effective over time
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