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366,367,368 For example, Nlgn1- knockout mice exhibit ASD-like repetitive behaviours and corticostriatal synaptic abnormalities, 369 whereas mice lacking Nlgn3 exhibit similar behavioural changes caused by neuronal inhibitory transmission from D1-MSN in the nucleus accumbens (NAc)

doi: 10.1002/ejlt.201800501

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Moreover, mice blood analysis revealed marked decreases in the population of T315I-Bcr-Abl-32D/EGFP + cells by the combination of JMF4073 with UK-5099 treatment (Fig

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This enzymatic defect reduces synthesis of glutathione and the resulting low intracellular glutathione levels remove the normal tonic inhibition of the enzyme -glutamylcysteine synthetase (Figure 1 and Figure 2, reaction 4)