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However, substrate-dependent CIII and CIV OCR showed a tendency, although not significant, to be increased in GSH-deficient FoB cells compared to control FoB (Fig
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The two target different systems: Semax works through neurotrophic signaling while NAD+ supports mitochondrial function and energy metabolism at the cellular level
Fry and Lohnes, 2010)
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Investigacin de referencia Ames et al., 2004 (PNAS): Bruce Ames y colaboradores del Buck Institute demostraron en ratas envejecidas que la combinacin de ALCAR (1.5% en agua) + cido alfa-lipoico (0.5% en dieta) restaur la funcin mitocondrial, redujo el estrs oxidativo mitocondrial y mejor el rendimiento cognitivo (laberinto de agua de Morris) hasta niveles cercanos a ratas jvenes
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