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This led to acquisition of resistance to high levels of exogenous gliotoxin (50 g/ml) (Figure 6C) thereby confirming the key role of gliT in protection against gliotoxin toxicity in gliotoxin-nave fungi

doi: 10.1016/j.bbi.2024.03.019 136

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Mitochondrial impairment serves as a key driver of ferroptosis by elevating reactive oxygen species and promoting the iron-mediated Fenton process, wherein ferrous ions facilitate the transformation of hydrogen peroxide into highly reactive hydroxyl radicals, thereby exacerbating oxidative damage and triggering ferroptotic cell death (121, 125, 126)

From gut to brain: Peripheral BPC-157 administration increased serotonin synthesis in substantia nigra, produced antidepressant effects in Porsolt tests, and counteracted serotonin syndrome behavioral manifestations

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