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In an APP/PS1 model of AD, D-Ala2-GIP reduced activation of chronic inflammatory response in the brain, oxidative stress, synapse loss, amyloid plaque burden, and DNA damage [106]

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This process represents the summary of the GSH-related aspects with regards to: (i) changes in cellular GSH redox homeostasis through decreased GSH/GSSG ratio, due to either GSH oxidation or active GSH export in relation to the initiation or execution of the apoptotic cascade

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Ottersen OP, Zhang N, Walberg F

And theres a bunch of theories of aging, but if you take them all, you can always ask the question, why theres like the information theory of aging, like, why is the information not being carried out correctly