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The insulin-like growth factor system and cancer

TRPM4 is activated by increased intracellular ATP concentration and oxidative stress, but it is inhibited by intracellular ATP depletion (Nilius et al., 2004

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Conversely, some patients develop subclinical or overt hypothyroidism as autoimmune thyroid disease evolves

Pharmacological restriction of cellular NADPH availability by targeting its synthesis pathways to impair NADPH homeostasis is currently recognized as a crucial and potential strategy for cancer treatment