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Methyl B12 is the most promising treatment for patients with severe neurological symptoms, bypassing several steps in the absorption cycle and helping to relieve or completely reverse symptoms

So the strongest neuroregenerative claims attributed to TB-500 are, strictly, claims about its parent protein component-level evidence, not direct evidence for the fragment

Minimal response may warrant increasing by 50-100mcg

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Pyridoxine appears to improve the behavioral side effects (discussed below)

That second pathway explains why prolonged deficiency causes neurological damage and not simply anaemia