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Adding to the complexity, if the viral glycoprotein cross-seeds an endogenous brain protein, years later even the viral protein need not be present, as the run-away proteopathy would now involve only endogenous brain protein (s)

There were several limitations of our work, the main one being that this was an in vitro study using human cell lines in 2D cell cultures

We're throwing all that potential data away that could be used to actually identify real safety and evidence signals

3), suggesting that ALT has been activated and is responsible for the increase in telomere length seen in the cancer cells