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Furthermore, the increased expression of HO-1 in individuals suffering from heart failure serves to mitigate the adverse effects of pathological left ventricular remodeling, diminish myocardial hypertrophy, minimize oxidative stress, and abate inflammatory activation [96]

doi: 10.1016/j.jocn.2020.09.054 201 MenziesFMFlemingARubinszteinDC

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