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The authors reported that high intracellular levels of the lactone caused apoptotic cell death by upregulation of COX-2 mRNA and protein, as well as increased formation of peroxisome proliferator activated receptor (PPAR)-activating PGD2

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Long-term, perhaps 25% of initial weight loss persists

CRE-treated animals indicated a significantly higher activity of GPx than exposed nonCRE- or CRE-AgNPtreated animals

Acknowledgments The authors express their thanks to Drs Eckhardt Ferdinandi and Claude Larose (formerly of Theratechnologies Inc.) for providing the GLP-1 receptors agonists labelled above as G001 and G002, to Olga Plaskon, Estera Karolczyk, Kasia Zajkowska and Gurpal Bansal for technical assistance, and to Professor Huw Jones, Middlesex University for help with statistical analyses

In the F1 generation, LC3 protein levels exhibited no significant variations between the treatment groups (D1 and D2) and the controls ( Figures 7AC )