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Treating RPE cells with the iron chelator deferoxamine (DFO) can lead to a reduction in intracellular iron ions (154), an increase in genes related to iron transport, a significant elevation in the level of hypoxia-inducible factor HIF2, and impairment of mitochondrial function, indicating that the RPE is the main target tissue for DFO toxicity, resulting in atrophy and degeneration of the RPE and posing a risk of retinal detachment

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10.3389/fmicb.2014.00223 158 WanZ.ZhangX.JiaX.QinY.SunN.XinJ.et al

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In models of high-fat diet-induced obesity, GLP-1R agonism has demonstrated the ability to enhance neurovascular coupling, improving the relationship between neuronal activity and blood flow in the brain

Detox diets may not be the best solution for a healthy mind and body