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Researchers are particularly interested in its behavior under metabolic stress, where mitochondrial-derived peptides appear to play a regulatory role in how cells adapt energy production to changing demands

The GHK sequence also appears within the alpha-2(I) chain of type I collagen, leading to the hypothesis that collagen degradation at wound sites releases GHK fragments that then bind copper and signal for new collagen production, creating a self-repair feedback loop

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This amplification cascades into increased BDNF-TrkB signaling, enhanced synaptic plasticity markers (PSD-95, synaptophysin), and upregulated dendritic spine density

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