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Research has shown that dioscin effectively reduces oxidative stress by scavenging reactive oxygen species (ROS) and boosting the activity of key antioxidant enzymes such as SOD, CAT, and glutathione peroxidase (GSH-Px)

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Proposed mechanisms for hepatocyte necrosis include extreme hypertrophy leading to reduced sinusoidal blood circulation, hypoxia, and necrosis (Slauson and Cooper 2001) and/or metabolic activation forming more toxic active metabolites (Farber 1980)

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Behav Brain Res 2017;317:461-9

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These compounds play an important role in preventing oxidative stress caused by the endogenous formation of hydrogen peroxide (H 2 O 2 ) by aerobic metabolism

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