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[71] also reported that the activation of STAT3 enhanced the FoxO-dependent expression of inhibitor of B (IB) proteins which induced the accumulation of NF-B complexes into cytoplasm, inhibiting the pro-inflammatory NF-B-dependent gene expression

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To test whether the observed p53 and apoptotic induction upon nutrient withdrawal is cell-autonomous, we employed in vitro adipocyte models

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