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(2003) Beta-arrestin-mediated activation of MAPK by inverse agonists reveals distinct active conformations for G protein-coupled receptors

However, for most agents, including warfarin and digoxin, overall systemic exposure is not clinically meaningful, and routine dose adjustments are generally unnecessary

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All the mechanistic evidence for its relevance to PMOS is extrapolated from pre-clinical data and its known anti-inflammatory mechanisms

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