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PMID 15465973

Thus, this study revises the conventional doctrine of GSH function in protein redox regulation and liver pathophysiology, indicating that hepatic GSH depletion by Chac1 overexpression may represent a potentially novel therapeutic approach for MASLD (Figure 6G)

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Dysfunction within the CNS Brain imaging studies have demonstrated an association between PDPN and structural changes in key somatosensory regions, such as the thalamus and somatosensory cortex, and altered functional connectivity within ascending pain pathways and cortical regions involved in pain processing