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Radiation-resistant NPC cells consistently displayed active up-regulation of CPT1A, and inhibition of CPT1A could render NPC cells once again vulnerable to radiation treatment by inducing mitochondrial apoptosis (Tan et al., 2018)

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These results indicate that AsGGT1, AsGGT2, and AsGGT3 are the functional GGT proteins with a preference for -glutamyl- S -allyl-L-cysteine as a -glutamyl donor substrate over -glutamyl- S -allyl-L-cysteine sulfoxide