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It achieves a much higher drug concentration in the mucosa with a more rapid onset of action and better efficacy than oral 5-ASA monotherapy or rectal steroids alone[25]

Figure 1 Gross pathological changes expected in this disease model included increased lung mass, increased cellularity of the bronchoalveolar lavage fluid (BALF), epithelial thickening, and pockets of immune cell infiltrations in the lung parenchyma

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& Bautista, J

The infectious organism is not implicated in SIBO, but the resulting disruption to physiology then sets the patient up for SIBO development, typically within a few months after their bout of gastroenteritis