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The endometrium is highly responsive to cyclic changes in the ovarian steroid hormones estradiol and progesterone, which largely exert their effects via their cognate nuclear receptors and downstream regulation of gene transcription within multiple cellular compartments of the endometrium ( FIGURE 1 Although both isoforms of the nuclear estrogen receptor (ER), ER-alpha and ER-beta, are expressed in all parenchymal endometrial cell types (including glandular epithelial and stromal) across all phases of the menstrual cycle, ER-alpha is the predominant form uniformly (Matsuzaki et al., 1999)

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