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The current hypothesis is that tyrosine crystalizes in corneal epithelial cells, disrupting their lysosomes and initiating an inflammatory response

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GM-CSF not only feeds back to antigen-presenting cells to sustain IL-23/Th17 responses [110] but also facilitates eosinophil recruitment and functional enhancement [111]

BPC 157 peptide also plays a role in inflammation regulation

The most intriguing aspect that is nowadays emerging is the crosstalk between ATG and apoptosis through a link with redox signaling governed by GSH

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