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Thus, this regulation by the HGF/c-Met pathway constitutes a protective mechanism in normal cells (Clavijo-Cornejo et al

In multiple preclinical models, pharmacological inhibition of ferroptosis preserves myocardial and vascular function in settings such as ischemic injury, heart failure, and atherosclerosis, positioning ferroptosis as both a central disease driver and a potential therapeutic target

[Google Scholar] 244.Iftikhar I.H., Schimmel M., Bender W., Swenson C., Amrol D

PMID: 23493030

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Thus, data in support of each hypothesis was derived from different animal models, with mitochondrial oxidative stress being critical for APAP hepatotoxicity in mice on a normal diet, without appreciable LPO generation