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MOTS-c improves mitochondrial function for improved energy use, while FTPP causes fat cell death

However, the endogenous and exogenous antioxidants can work synergistically to maintain or establish a redox balance

3.3 Oxidative stress and mitochondrial dysfunction EED-induced oxidative stress represents a central mechanism underlying reproductive toxicity, with mitochondrial dysfunction serving as both cause and consequence of excessive reactive oxygen species production

While DSIP functions as a broad neuromodulator within central nervous system signaling pathways, Epitalon and Orexin represent more targeted models of circadian regulation, cellular aging processes, and arousal signaling