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Targeting of a CCK2 receptor splice variant with 111In-labelled cholecystokinin-8 (CCK8) and 111In-labelled minigastrin

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Indeed, IDH1 inhibition or glutamine starvation in MYC-overexpressing cells decreased KG abundance (Extended Data Fig

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Although the mechanism explaining why C-KIT is pathogenetically important in sun-protected melanomas remains poorly understood, it does not prevent the protein from being a potentially effective therapeutic target and a series of C-KIT inhibitors are currently under pre-clinical and clinical investigations (82, 8789)