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Because the beta-oxidation of fatty acids requires substantial amounts of oxygen to proceed, this hypoxic environment further stalls the mitochondrial engines
The complex is C14H24CuN6O4 (glycyl-L-histidyl-L-lysine + one Cu ion in a square-planar coordination)
In conclusion, when confronting drug resistance related to DNA repair, it is essential to focus on the specific DNA repair pathways implicated and the mechanisms by which therapeutic agents operate
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However, prolonged ischemia may impair autophagy, leading to ineffective clearance of damaged mitochondria [198]
