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Mitochondrial dysfunction has been recently proposed as a key pathophysiological mechanism [3] linked to processes commonly found in depression, including neurotoxicity, impaired neuroplasticity, inflammation and insulin resistance [2,3,4,5]

Gut microbiota modulates osteoclast glutathione synthesis and mitochondrial biogenesis in mice subjected to ovariectomy

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Within these pathways, phospholipase C (PLC) is speculated to play a critical role by potentially catalyzing the formation of second messengers, such as inositol triphosphate (IP3) and diacylglycerol (DAG)

Individuals with high-risk scores, persistent abnormal liver function tests for more than six months, or clinical signs of advanced liver disease should be referred to a hepatologist

Diet-gut microbiota interactions on cardiovascular disease