Thus, future studies should include other inflammation-related biomarkers which may explain part of the variance in the clinical neuropsychiatric symptoms, including autoimune responses, the cytokine network, the NLRP3 inflammasome, and hypernitrosylation
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The second runs through RAS, RAF, MEK, and ERK1/2, which control cell-cycle entry and satellite-cell proliferation [5]
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Persistent or worsening reactions should be reported to your provider