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It is increasingly clear that fibrosis resolution, and possibly regeneration, can be coaxed out of mammalian tissues by disrupting profibrotic mechanosignaling, and by eliminating, inhibiting, or manipulating myofibroblast activity directly or by several upstream interventions 311,313

All trials had a parallel-group design and 15 were open-label (Table 1)

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However, compared with native GLP-1, which is rapidly degraded and has a short half-life of approximately 2 min, semaglutide has been structurally modified by the addition of a hydrophilic spacer and a fatty acid side chain, allowing reversible binding to albumin [36]

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