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We intend to specifically investigate the redox control of GSAAT, analyze the redox-dependent shifts in the thiol-disulphide state of GSAAT, and identify the redox-dependent cysteines responsible for changes in the structure, enzymatic activity and stability of the protein

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The mechanism likely involves GLP-1 receptors found directly in joint tissue, with liraglutide shown to reduce matrix metalloproteinases (enzymes that break down cartilage) and shift macrophages in the joint to an anti-inflammatory state [19]

Diabetes Metab 1990

The microtiter plate provided in this kit has been pre-coated with an antibody specific to Guinea pig TNFa

Read more about this FDA announcement here