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Similarly, hearts from transgenic mice overexpressing GPx1 had preserved mitochondrial and cardiac function in the presence of doxorubicin, with increased efficiency of mitochondrial respiration and better contractility and diastolic function compared wild type hearts exposed to doxorubicin [44]

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In accordance with this, studies where iron accumulation was forced in tumor cells, either by overexpression of TFR1 or through iron-rich nutrition, demonstrated enhanced tumor growth [65, 66]