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Another potential mechanism suggested by experimental research is the potential GHK-Cu action on synthesis of matrix metalloproteinase-2 (MMP-2) and tissue inhibitors of metalloproteinases (TIMPs), which are deemed crucial for extracellular matrix remodeling
Poldip2 mediates blood-brain barrier disruption and cerebral edema by inducing AQP4 polarity loss in mouse bacterial meningitis model
Extracellular vesicles and chronic inflammation during HIV infection
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