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doi: 10.1016/j.immuni.2015.01.011 91 EmgardJKammounHGarcia-CassaniBChesneJParigiSMJacobJMet al

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Pathogenesis Although not fully understood, the pathogenesis is believed to involve the strengthening of collagen as a result of reactions associated with advanced glycosylation end products or a buildup of sugar alcohols in the upper dermis (37,38)

Although research is ongoing, early evidence suggests that KPV can: Reduce inflammation within the GI tract

- GHK-Cu is a copper-binding peptide studied in advanced research models involving cellular signaling, tissue remodeling, collagen-related pathways, oxidative stress, and skin/hair-focused research applications

Under normal circumstances it is produced by our bodies