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The data presented here suggest that Nlel-AnglV and Dihexa increase miniature synaptic activity by increasing dendritic spine densities and altering the morphological phenotype of postsynaptic spines in- vitro and may represent the mechanism that underlies facilitated learning observed AnglV analogues

Random-effects model was applied due to the high heterogeneity among the studies (I-squared = 100%)

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Peptide analogs that mimic GHK-Cu but resist enzymatic breakdown

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