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The repair function of APE1 is protective by switching on antioxidant and cell survival mechanisms after oxidative DNA damage to neurons (Jiang et al., 2009)

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In: Chawla S, Singh S, Husen A, editors

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Interestingly, we found increased interstitial fibrosis and vascular abnormalities consistent with inflammation and fibrosis in coronary and aortic sections from older GPx1 +/ mice [39], illustrating the detrimental effects of chronic oxidative stress on the vasculature