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Several viruses, including IAV and HIV-1, utilize mitochondrial apoptosis to facilitate viral spread, although some delay apoptosis early in infection to optimize replication (Galluzzi et al., 2008)

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However, owing to the lack of consideration for brain capillary binding or sequestration, the reliability of these results regarding brain uptake pharmacokinetics has been questioned [21] as well as whether results could be translated into humans