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In healthy tissue, the peptide maintains NO homeostasis by modestly increasing endothelial NOS (eNOS) for beneficial vasodilation while suppressing inducible NOS (iNOS) that drives pathological inflammation

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But in the three- and four-week-old mice the densities were significantly reduced in the DG-NR1KO mice (three weeks: p = 0.0075, t (13) = 3.159, n = 7 control and 8 DG-NR1KO mice, four weeks: p = 0.0048, t (13) = 3.394, n = 7 control and 8 DG-NR1KO mice, independent-sample t tests, two-tailed)