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Metabolic research has established a correlation between hyperuricemia and disruptions in the primary bile acid pathway or intestinal metabolism, suggested that targeting the gut microbiome could offer innovative therapeutic approaches for managing hyperuricemia and its associated complications

GSH produced in the cytosol can be transported directly to other cellular organelles by glutathione transporters [7]

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The inhibitory effect was dose-dependent and started already at 2.5 M SFN