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In microglial BV2 cells semaglutide dose-dependently inhibited apoptosis induced by -amyloid (1-42) and promoted the transformation of microglia from M1 to M2, thereby exerting anti-inflammatory and neuroprotective effects [50]

The level of IL-6 in liver tissue was significantly higher (P 0.05)

Because the follow-up time was fairly short, these results may have been caused by reverse causality, where a developing, undiagnosed condition was influencing IGF-1 levels at the time when the samples were taken

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Most people only see the headline, not the financial reality behind these negotiations