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In doing so, it promotes retention of metallothionein-bound copper in the enterocyte that will then be excreted in the feces when the enterocyte is shed.11 It is on this basis that zinc was postulated to reduce intestinal copper absorption and became a modality of treatment for Wilsons

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C.ZeiglerA

For example, intradiscal injection of bone morphogenetic protein-2 (BMP-2) induced degenerative changes [92] and weekly intradiscal treatment with growth and differentiation factor-5 (GDF-5) or insulin-like growth factor-1 (IGF-1) resulted in inflammatory reactions in the adjacent vertebrae and connective tissue infiltrates in the NP, causing the IVD to collapse [93]

Shiroky, J