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Because Dihexa activates HGF/c-Met signaling , and this pathway is also involved in some types of tumor progression, caution is advised in: Patients with active cancers History of certain malignancies No human data confirms a cancer risk, but Dihexa is best avoided in patients with cancer unless under strict clinical supervision

Bioinformatics methodologies indicate an overexpression of AKR1C3, accompanied by a downregulation of AKR1D1, correlating with unfavorable prognosis and a shortened median survival time in HCC

This helps identify patterns and allows for early intervention if side effects become concerning

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These findings are most comparable to populations with metabolic disease in healthcare systems with similar access and registration of suicides