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Using metabolomic analysis, the discrepancies observed between the lipid profiles of keratinocytes after UVB exposure were found to originate mainly from oxidized PE, and the accumulation of the oxidized PE is believed to be essential for executing keratinocyte ferroptotic cell death after UVB radiation

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This therapy has gained attention for its ability to aid in injury repair, especially in athletes or individuals dealing with joint, muscle, or tendon injuries

This deficiency was associated with an increase in proteins involved in myelin clearance proteins as well as promoters of axon growth

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