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C-Met expression is controlled by the -catenin signaling and downregulated by inhibition of Wnt signaling using Frzb and DNLRP5, two Wnt antagonists [103, 104]

A "natural" origin of a substance tells you nothing about overall safety at a synthetic dose, by an unphysiological route, in a manufactured form whose purity is not independently verified

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GHK-Cu functions as both a signal peptide (triggering collagen synthesis) and a carrier peptide (delivering copper)

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