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The ability of glucoraphanin-derived sulforaphane to reduce oxidative stress in different settings is linked to activation of the nuclear factor E2-related factor 2 (Nrf2)-dependent pathway (see Biological Activities)

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However, to clearly understand the mechanisms and level of toxicity of sALS astrocytes, it remains to be investigated if sALS and fALS astrocytes are equally detrimental to motor neurons upon activation [in response to an ALS environment or to lipopolysaccharide (LPS)] and their secretion pattern in such a context