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For instance, ACSL4 promotes the incorporation of PUFAs into phospholipids, making them vulnerable to lipoxygenase (ALOX)-mediated oxidation, which compromises the lipid bilayer and induces ferroptosis [17, 18]

Fluorescence imaging confirmed effective uptake and targeted intracellular localization of DNZ-NLPs, with FITC and DAPI signals showing partial overlap, suggesting nuclear or perinuclear targeting

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Abstract Objectives: Hepatotoxicity is a severe outcome of methotrexate (MTX) therapy, limiting its clinical use and contributing to its related morbidity and mortality