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GHK-Cu has been studied for its potential relevance to connective tissue and extracellular matrix regulation in cell and tissue models, while TB-500 and BPC-157 have been investigated in preclinical systems involving soft tissue, tendon and ligament models, and angiogenesis related signaling
Vendors often use the names interchangeably, or confuse the two
While the mechanism of action for BPC-157 is still unclear, it is believed to entail the upregulation of growth factors, the modulation of nitric oxide (NO) synthesis, and the control of functions of collagen fragments that are associated with bone morphogenic proteins [1]
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