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(2013) suggest that the lack of significance is likely due to the high levels of cointernalization under control conditions

doi: 10.1152/physrev.00031.2024

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PloS one 7 , e42529, doi:10.1371/journal.pone.0042529 (2012)

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Under lipotoxic conditions, excessive peroxisomal substrate flux may itself become a source of oxidant stress, while inadequate catalase-dependent detoxification can facilitate ROS spillover toward mitochondria, thereby aggravating membrane depolarization, inflammasome signaling, and bioenergetic collapse ( PPAR- is particularly important in regulating the activity of the antioxidant enzymes catalase and HO-1