366,367,368 For example, Nlgn1- knockout mice exhibit ASD-like repetitive behaviours and corticostriatal synaptic abnormalities, 369 whereas mice lacking Nlgn3 exhibit similar behavioural changes caused by neuronal inhibitory transmission from D1-MSN in the nucleus accumbens (NAc)
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Transitional changes in the structure of C-reactive protein create highly pro-inflammatory molecules: Therapeutic implications for cardiovascular diseases
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All measurements were performed in 5-replicates
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