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Figure 4 The involvement of PI hydrolysis/Ca 2+ and cAMP/PKA pathways in OA-mediated GLP-1 and PYY release was examined using selective inhibitors

Our nanoBRET findings demonstrate that DA activation of the 2A -adrenergic receptor primarily results in recruitment of G i1 to the receptor with negligible -arrestin2 recruitment while NE treatment causes robust recruitment of both the G protein and -arrestin2