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Employing gene editing techniques alongside both in vivo and in vitro experiments, we provide novel evidence that the dysfunction of SLC25A39-mediated mitochondrial import of GSH constitutes a pathological alteration following TBI, leading to mitochondrial dysfunction and subsequent initiation of oxidative stress and neuronal cell death
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Strategies to minimize reactions include rotating injection sites consistently to avoid repeated trauma to the same area, ensuring the solution is at room temperature before injection (cold injections can increase irritation), injecting slowly rather than quickly, and using a smaller gauge needle for less tissue trauma
[DOI] [PubMed] [Google Scholar] 156.Yang J.L., Chen W.Y., Chen Y.P., Kuo C.Y., Chen S.D
